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In the search for a substance that fit the criteria mentioned, the crystal structure of imatinib bound to Abl was examined.
Even the conversion rate from inoperability to operability with neoadjuvant therapy is ill-defined.
Response rates were higher with dasatinib therapy, although overall PFS and OS rates were not significantly higher in the dasatinib arm.
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Our data show that imatinib resistance may occur on the basis of clonal selection of cells which reactivate BCR-ABL despite continuation of imatinib therapy.
The patient was discharged home on intravenous antibiotics and was stable after two weeks.
In a 1998 case series of 50 patients, treatment with a combination of anticoagulation, steroids and plasmapheresis or IV GG resulted in a 70% survival rate.
Used as maintenance therapy, it is useful for the prevention of manic episodes, but is not useful for bipolar depression.
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It works by blocking the action of an abnormal protein that signals cancer cells to multiply.
Patients who have a rapid decrease in the number of cells with the Philadelphia chromosome by three months after starting treatment may have the best long-term outcomes.
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Severe CHF and left ventricular dysfunction has been reported, mostly in patients with comorbidity and risk factors, including advanced age and history of cardiac disease.
We next assessed the effect of imatinib on VEGF mRNA and protein expression in three neuroblastoma cell lines.
La LMA invade tejidos no hematopoyéticos como meninges, parénquima cerebral, testículos u ovarios, o piel (cutis leucémico).
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The mainstay of therapy, anticoagulation, was delayed in this patient until she was safely past the immediate post-operative period and pulmonary hemorrhage had subsided.
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Los resultados de un metanálisis de siete estudios publicados en pacientes de APL indican que la combinación de trióxido de arsénico y ATRA puede ser más eficaz que el trióxido de arsénico solo en inducir una RC.
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Surprisingly, the researchers noted that advancedstage colon cancer patients only appeared to derive benefit from moderate not vigorous activity.
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Imatinib, a rationally designed tyrosine kinase inhibitor, has shown activity against leukemic cells in vitro and in vivo.
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Compared to traditional methods that have allowed us to investigate human cells in culture in twodimensions, these cultures let us investigate the threedimensional structure and function of the cells as they are developing, giving us more information than we would get with a traditional cell culture.
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Despite, or even because of this, ponatinib is a potent drug and targets not just most of the known mutations on the Bcr-Abl TK but, most importantly of all, T315I.
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After the mutation and hyperactive bcr-abl protein were discovered, the investigators screened chemical libraries to find a drug that would inhibit that protein.
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The reasons for this are the higher rates of depression and SSRI use, decreased kidney function, and polypharmacy, especially the concomitant use of thiazide diuretics and neuroleptics.
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The Kaplan-Meier product-limit method was used to estimate time to and duration of response, progression-free survival, and overall survival.
In addition to therapy delivered directly to the brain and spinal fluid, systemically administered agents are also an important component of effective CNS prophylaxis.
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Because disease progression has been reported to follow the cessation of imatinib therapy, patients with unresectable or metastatic disease are often treated with a TKI indefinitely, as long as the disease does not progress and patient tolerance permits.
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Another important mechanism by which imatinib ameliorated the disease was by its action on antigen presenting cells (APC).
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Imatinib mesylate, a tyrosine kinase inhibitor, is a first-line chemotherapy agent used in the treatment of CML.
Fluid Retention Associated with Imatinib Treatment in Patients with Gastrointestinal Stromal Tumor: Quantitative Radiologic Assessment and Implications for Management.
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Chronic necrotizing pulmonary aspergillosis (CNPA), also known as semi-invasive or chronic granulomatous aspergillosis, was first described in 1981.
On current clinical trials, many patients who receive prophylactic or presymptomatic cranial radiation are treated with an even lower dose.
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This analysis reported elevated rates of PAOD for nilotinib as compared with imatinib by screening with on-treatment ABI and showed an elevation of both cholesterol and LDL in nilotinib-treated patients.
Doses as low as 81mg per day a very common dose for individuals with cardiovascular ailments have been shown effective.
imatinib was withheld and then restarted at 300 mg qd in arm a and at 300 mg bid in arm b
This large, prospective, intention-to-treat, randomized study on therapy for high risk patients with CML failed to demonstrate a benefit of imatinib administered at a higher dose for the primary end point, as well as for any other measure of efficacy, toxicity, and compliance.
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Because 18FDG-PET imaging may detect the activity of imatinib in GIST much earlier than CT imaging, imaging of GIST with 18FDG-PET may represent a useful diagnostic modality for the very early assessment of response to imatinib therapy; a decrease in tumor avidity for 18FDG may be detected as early as 24 hours after a single dose of imatinib.
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The abbreviations used are: CML, chronic myelogenous leukemia; Ph, Philadelphia; CP, chronic phase; AP, accelerated phase; CE, clonal evolution; CR, complete response.
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